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Tricresyl phosphate

TEL: 13816217984

Specifications:

Product details

English Name Tricresyl phosphate
CAS NO. 1330-78-5
Molecular Formula C21H21O4P
Son 368
Description Appearance: CLear liquid
Flash point ℃: ≥225℃
Acid value mgKOH/g: ≤0.1
Relative density g/cm after (20°C):1.165-1.180
Chroma (pt-co) no: ≤80
Application This product is colorless or light yellow transparent oily liquid. Odorless, strong stability, not volatile, with good plasticizing Flame retardant, oil resistance, electrical insulation, easy to process. Insoluble in water, soluble in benzene, alcohol, ether, vegetable oil, mineral oil and other organic solvents. This product is an important oF vinyl resin and nitrocellulose plasticizer, used in the paint Film can increase the Flexibility, it has excellent intermiscibility, synthetic rubber, PVC, polyester, polyoleFin and soFt polyurethane Foam plastics Flame retardants, electronic potting glue can also be used For polyurethane. This product can give high polymer good abrasion resistance, weatherability, mildew resistance, radiation resistance and electric properties. This product adopts the new craFt improvement but becomes, belongs to the environmental protection product, in the production process basically does not have three wastes to produce, and the product low poison.
Appearance: CLear liquid Smell: aroma
Flame retardant: not flammable melting point (℃):  -33
Boiling point (℃): 420 Natural temperature (℃):   385
Saturated vapor pressure (KPa):  1.33 (265℃) Steam density (gas=1):12.7
relative density:1.16 Solubility: insoluble in water, soluble in organic solvents such as alcohol, benzene.
Package 230kg/drum

Health hazards: This product has an inhibitory effect on pseudocholinesterase in the body, but does not inhibit true cholinesterase. The ortho isomer (TOCP) is mainly responsible for delayed toxic neuropathy, while the meta - and para isomers are actually non-toxic and do not cause neuropathy.

Acute poisoning: When taken orally in large quantities, nausea, vomiting, and diarrhea first appear, which subside in about 1-2 days. Delayed onset neuropathy usually occurs after 7-28 days, mainly in the motor type. It often begins with severe spasmodic pain in the gastrocnemius muscle, which may be accompanied by mild sensory abnormalities in the lower limbs. Later, there may be weakness in the lower limbs, which rapidly progresses to obvious flaccid paralysis. In severe cases, pyramidal tract sign may also appear. The recovery speed of neurological symptoms and signs varies significantly among individuals. It can also be absorbed through the skin and respiratory tract.

Chronic poisoning: Long term exposure to small amounts of adjacent triphenylphosphate can cause neurological damage similar to acute poisoning.

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